60 Arboviruses
James Whitehorn 04/10/18
Arboviruses are arthropod borne viruses
60.1 Dengue
NS1 antigen test
Can be self limiting with febrile illness. Can be severe with DIC, acidosis, death.
Transmitted by aedes
21,000 deaths. 2 million severe cases. Up to 500 million infections. (Globally yearly). This makes it the most abundant arboviral infection globally.
Can either be endemic/epidemic. It can overwhelm health systems during epidemic period (the rainy season)
60.1.1 Definitions
Reclassified in 2009 into:
- Dengue
- Dengue With Warning Signs
- Severe Dengue
We don’t talk about Dengue Haemorrhagic Fever any more
Warning Signs:
- Abdo pain
- Vomiting
- Fluid
- Mucosal bleed
- Lethargy
- Hepatomegaly
- Increase in HCT
Severe Dengue:
- Shock
- Resp Distress from Pulm Oedema
- Severe Bleeding
- Severe Organ (liver, brain, heart) Involvement
60.1.2 Virus
Dengue Virus (Flavivirus - 4 serotypes). Basically 4 totally seperate viruses
Single stranded RNA virus
Simple virus genome. We use one of the non structural proteins in the genome for the diagnostic test.
60.1.3 Illness
- Febrile Phase
- Critical Phase - when you get plasma leak and properly sick
- Recovery Phase
When monitoring the disease progression, we use the platelet count as a measure of severity, at it’s lowest is usually the sickest point. So when platelets are improving, patient is likely to improve
Incubation is between 4-14 days.
Most cases self limiting but fairly debilitating fevrile illness. With fever, headache, rashes
Rash: Petechial
Then in recovery you get a weird islands of white in a see of rash, like a sunburn
60.1.4 Platelets
Low in all patients w/ dengue
Normal platelets = no dengue
60.1.5 Severe
- Vascular Depletion (increased vascular permeability)
- Haemorrhage
- Organ Impairment
Liver - LFTS almost always raised. Can get severe hepatitis
Brain - Acute encephalitis. more common in kids
Heart - Myocarditis, arrhythmias (bradycardia, others), myocardial depression
60.1.5.1 RF for severe disease
Why are secondary infections more likely to be severe?
Probs due to antibody dependent enhancement
This is where preexisting antibodies to old infection bind to a virus, but dont knock it out properly, the antibodies pull it into a monocyte (who have FC gamma receptor cells), and the virus then replicates in the cell, increasing viral load rather than lowering it!
Infants are probably the most vulnerable groups to severe disease, due to having maternal antibodies causing this response
60.1.5.2 Other RF’s
- Older Ages
- Under 5’s
- Pregnant Women
- Comorbidities (DM/HTN)
- Obesity/Malnutrition
60.1.6 Capillary Leakage
Very transient but very severe.
There’s no lasting damage to the endothelial layer. This is prob caused by the NS1 antigen.
If we block this antigen, we can maybe stop the leakage
60.1.7 Diagnosis
- NS1 Rapid Test
A really good rapid dest for most serotypes of dengue
A week later you can use serology
60.1.8 Treatment
Supportive, fluids, antipyretics.
Most patients could be managed as outpatients, but admit if warning signs.
Need careful fluid resuscitation in dengue. Recommend reserving colloids for refractory shock.
Really gentle fluids. CHECK ALGORITHM FROM SLIDES
60.1.8.1 Therapeutics for dengue?
We’re talking about therapeautics to prevent vascular leakage phase.
No viable drugs for it.
60.1.9 Vaccine?
Dengvaxia was the candidate
Live attenuated. All 4 serotypes. Uses a yellow fever back bone.
There are major safety concerns. Give it to people who haven’t had dengue before, you’ll increase their risk of severe disease
So now it’s withdrawn in many places
60.1.10 Vector Control
Thinking about introducing artifical wolbachia to aedes aegypti. May prevent it transmitting dengue.
60.2 Chikungunya
Chikungunya - “that which bends up” - refers to the bone pain caused
Alphavirus (another arbovirus)
Only one serotype, so once you’ve had it, you’re immune
Clinically similar to dengue, but you don’t get shock, and you only get minor bleeding
Aedes albopictus (asian tiger mosquito)
Most infections are symptomatic (loads of dengue aren’t)
Loads of polyarthralgia and skin manifestations
You can get some severe organ involvement, but that is really rare.
20-80% of patients get persistant joint symptoms (up to three months). And half of those will get a secondary inflammatory arthritis (needed treated w things like methotrexate)
Diagnose w/ serology or PCR
All symptomatic management.
60.3 Zika
Another flavivirus, v close to dengue.
First seen in africa in the 1940’s. 6% seroprevalence back in the 1950’s in Uganda. We think there’s been a really recent mutation making it more severe.
Aedes africanas in african lineage.
Aedes aegypti in asian lineage (maybe also aedes albopictus). This is the one that’s big and important in the world now.
Only 1/5 of patients are symptomatic.
Fever/arthralgia/skin rashes. V mild disease itself. It’s the complications that are scary. An explosive outbreak in brazil in 2015, showed a load of microcephaly, and neurological stuff.
We think there’s zika perinatal transmission in early pregnancy.
60.3.1 Diagnosis
- Serology
- PCR
DOn’t test asymptomatic not pregnant patients. Early illness, PCR on blod. Later illness (1 week) PCR on urine/serology. Later still (after 2 weeks) serology
60.3.2 WHO
WHO guidelines suggest if you leave an endemic area wait 6/12 months before unprotected sex.
CDC say if asymptomatic wait 8 weeks.
60.4 Yellow Fever
Yellow fever is the namesake for the flavivirus family
Vaccine preventable
Aedes mosquitoes
600 million in at risk areas. Africa and Latin America. No one really knows why it hasn’t spread to asia, the mosquitoes live there.
In Latin America - sylvatic cycle In Africa - more urban human to human.
You have a:
- Infection Phase
- Remission Phase after (can also have subconjunctival haemorrhage and relative bradycardia)
- But some people get Intoxication with severe illness after
Diagnose:
- AST more than ALT (cardiac disease)
- Virus isolation
Severe yellow fever needs supportive treatment in ICU. Need isolated from mosquitoes.
Vaccine is live attenuated (17D vaccine), been around since 1930’s - but risks of viscerotropic disease in older age groups.